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020 _a9783319208190
024 7 _a10.1007/978-3-319-20819-0
_2doi
040 _aES-MaUEC
_bspa
050 4 _aQL757
_b.P384 2015
245 1 0 _aPathogen-Host Interactions :
_bAntigenic Variation v. Somatic Adaptations
_cedited by Ellen Hsu, Louis Du Pasquier.
250 _a1st ed. 2015.
260 _aCham, Switzerland
_bSpringer
_c2015
300 _a1 recurso en línea (VIII, 336 páginas)
_b35 ilustraciones, 26 ilustraciones en color
336 _aTexto
_btxt
_2rdacontent
337 _aelectrónico
_bc
_2rdamedia
338 _arecurso electrónico
_bcr
_2rdacarrier
490 0 _aResults and Problems in Cell Differentiation
_x0080-1844
_v57
505 0 _aEvolution of immunity and pathogens -- A host-pathogen interaction reduced to first principles: Antigenic variation in T. brucei -- Antigenic variation in Plasmodium falciparum -- Polymorphic mucin-like proteins in Schistosoma mansoni, a variable antigen and a key component of the compatibility between the schistosome and its snail host -- Fibrinogen-related proteins (FREP) in mollusks -- Somatic and germline diversification of a putative immunoreceptor within one phylum: Dscam in arthropods -- An immune effector system in the Protochordate gut sheds light on fundamental aspects of vertebrate immunity -- Variable lymphocyte receptors: a current view -- Antibody repertoire in fish -- Unique features of fish immune repertoires: particularities of the adaptive immunity within the largest group of vertebrates -- The evolution and structure of atypical T cell receptors -- Diversification of the primary antibody repertoire by AID-mediated gene conversion -- Antibody isotype switching in vertebrates.
520 3 _aThis volume provides in-depth reviews of model systems that exemplify the arms race in host-pathogen interactions. Somatic adaptations are responsible for the individualization of biological responses to the environment, and the continual struggle between host immune systems and invading pathogens has given rise to corresponding processes that produce molecular variation. Whether in mollusks or human beings, various host somatic mechanisms have evolved independently, providing responses to counter rapidly-changing pathogens. The pathways they utilize can include non-heritable changes involving RNA and post-translational modifications, or changes that produce somatic DNA recombination and mutation. For infectious organisms such as protozoans and flatworms, antigenic variation is central to their survival strategy. Evolving the ability to evade the host immune system not only increases their chances of survival but is also necessary for successful re-infection within the host population.
650 0 _9139394
_aParasitología
650 7 _aCitología
_2embne
_9139477
650 7 _aInmunología
_2embne
_9138330
700 1 _aHsu, Ellen
_eeditor literario
_993932
_0Local
700 1 _aDu Pasquier, Louis
_eeditor literario
_993933
_0Local
710 2 _aSpringerLink (Online service)
_0Local
_9106996
856 4 0 _uhttps://go.openathens.net/redirector/universidadeuropea.es?url=https://link.springer.com/book/10.1007/978-3-319-20819-0
_zAcceso a este recurso digital (usuarios Universidad Europea de Madrid)
907 _a.b12900709
_b10-10-17
_c18-01-16
942 _2lcc
_cLE
945 _aQL757 .P384 2015 EB
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988 _aEBOOK, EBSPRINGER, asignarmaterias_11febrero
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