000			06930nam a2200349 c 4500
988			_aSpringer_BiomedLife_2019
999			_c115554 _d115554 _x1
001			115554
003			ESmaUEC
005			20230110040236.0
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007			cr nn nnnaamaa
008			190617s2019 sz a o |||| 0|eng d
020			_a9783034806206
024	7		_a10.1007/978-3-0348-0620-6 _2doi
040			_aES-MaUEC _bspa _cES-MaUEC _dES-MaUEC
050		4	_aRB131 _b2019 EB
245	0	0	_aCompendium of Inflammatory Diseases _cedited by Michael J. Parnham.
264		1	_aBasel _bSpringer Basel _c2019
300			_a1 recurso en línea (1600 páginas)
336			_aTexto _btxt _2rdacontent
337			_2rdamedia _aelectrónico _bc
338			_2rdacarrier _arecurso electrónico _bcr
347			_atext file _bPDF
490	0		_aBiomedical and Life Sciences (Springer-11642)
505	0		_aAdhesion molecules of neutrophils -- Anaphylaxis (Immediate hypersensitivity): from old to new mechanisms -- Antiviral Responses -- Autophagy and Inflammation -- Basophils -- Cell signalling in neutrophils -- Co-stimulatory Receptors -- Dendritic Cells -- GILZ-Related Regulation of Inflammation -- Glial Cells -- Immunoglobulin receptors and inflammation.-Inflammation and autoimmunity -- Inflammatory Bowel Disease Models in Animals -- Leukocyte Recruitment -- Lymphocyte homing and trafficking -- Macrophage heterogeneity during inflammation -- Mast Cells -- Mechanisms of Macrophage Migration in 3-Dimensional Environments -- Microvascular Responses to Inflammation -- Modulation of inflammation by key nutrials -- Natural Killer Cells -- Neutrophil Extracellular Traps -- Neutrophil oxidative burst -- Obesity and Inflammation -- Osteoclasts in Inflammation -- Parasitism: A Unique Lifestyle Reflecting Dynamic Parasite- and Host(s)-Co-driven Processes -- Platelet, Endothelium, and Inflammation -- Regulatory T cells -- Sepsis Models in Animals -- Skin inflammation models in animals -- Th17 Response -- Toll-Like Receptors -- Angiogenesis Inhibitors -- Anti-asthma Drugs, Overview -- Anti-histamines -- Anti-muscarinics -- Antibiotics as anti-inflammatory drugs -- Antimalarial Drugs -- Azathioprine/Thiopurines -- Beta2 Agonists -- CD 20 Inhibitors - Rituximab -- CD80/86 Inhibitors -- Chromones - Cromoglycate and Nedocromil -- Colony stimulating factor therapeutics -- Corticosteroids -- Coxibs -- Disease-Modifying Anti-Rheumatic Drugs -- Fenamates -- Flavonoids as anti-inflammatory agents -- Gold Complexes -- Interleukin 1 Inhibitors -- Interleukin 12/23 Inhibitors: Ustekinumab Interleukin-6 Inhibitor - Tocilizumab -- Leflunomide -- Medicinal Fatty Acids -- Methotrexate.-NO-donating anti-inflammatory drugs -- Non-steroidal anti-inflammatory drugs -- Phosphodiesterase-4 inhibitors -- Propionic Acid Derivative Drugs (Profens) -- Salicylates -- Sulfasalazine and Related Drugs -- Tetracyclines -- Theophylline -- TNF Inhibitors -- Acute exacerbations of airway inflammation -- Allergic Disorders -- Alzheimer's Disease -- Antiphospholipid antibody syndrome -- Asthma -- Atopic dermatitis -- Autoinflammatory syndromes -- Behcet's disease -- Bronchiolitis obliterans -- Cancer and inflammation -- Cogan's syndrome -- COPD -- Diabetes type I -- Diabetes type II -- Diffuse panbronchiolitis -- Gaucher's disease -- Genetic susceptibility to inflammatory diseases -- Gout -- Graft vs Host Disease -- Granulomatosis with Polyangiitis (GPA) -- Grave's disease.-Guillaine-Barre disease -- Henoch-Schönlein Purpura -- Idiopathic Thrombocytopenic Purpura -- Inflammatory bowel disease -- Juvenile idiopathic arthritis -- Kawasaki Disease -- Lyme disease -- Osteoarthritis.-Osteoarthritis Genetics -- Polymyositis and Dermatomyositis -- Rheumatic Fever -- Rheumatoid arthritis -- Role of Cell Therapy in Autoimmune Disease -- Sarcoidosis -- Sepsis -- Spondylarthropathies -- ACKR3 -- Adhesion molecules -- Alarmins -- Antibacterial Host .-Defense Peptides -- Bacterial Lipopolysaccharide.-Biofilm -- Bone Morphogenetic Proteins in Inflammation -- C5a Receptors -- CCL18 -- CCR1 -- Ceramide 1-Phosphate: A Mediator of Inflammatory Responses -- Complement System -- CXCL4 und CXCL4L1 -- CXCR1 and CXCR2 and Ligands -- CXCR3 and its Ligands -- Cytokines -- Cytolytic Granules -- Foxp3 -- Heat Shock Proteins -- Histamine -- IFNgamma -- IkappaB -- Inflammasomes -- Interleukin 1 -- Interleukin 10 -- Interleukin 12 -- Interleukin 17 -- Interleukin 18 -- Interleukin 18 Binding Protein -- Interleukin 2 -- Interleukin 22 -- Interleukin 23 -- Interleukin 27 -- Interleukin 32 -- Interleukin 36 Cytokines -- Interleukin 4 and the related cytokines (Interleukin 5 and Interleukin 13) -- Interleukin 6 -- Interleukin 9 -- Interleukin-33 -- Janus Kinases (JAKs)/STAT Pathway -- Kinins -- Leukotrienes -- Mammalian Target of Rapamycin (mTOR) -- MAP Kinase Pathways -- NFkappaB -- Nitric oxide -- Nuclear Receptor Signaling in the Control of Inflammation -- Pathogen-Associated Molecular Patterns (PAMPs) -- Pentraxins -- Prostanoids -- Protease-Activated Receptors -- Reactive oxygen species -- Substance P in Inflammation -- TGF-b superfamily cytokine MIC-1/GDF15 in health and inflammatory diseases -- TNFalpha -- Type I Interferons.
520	3		_aInflammation has become one of the most exciting and rewarding areas of medical research. Recent years have seen a revolution in our understanding of how blood and tissue cells interact and of the intracellular mechanisms controlling their activation. This has revealed the underlying inflammatory pathology of many diseases and provided multiple new targets for anti-inflammatory and immunomodulatory therapy. The Encyclopedia of Inflammatory Diseases will cover the following areas: Inflammatory Processes and Cells Inflammatory Diseases Mediators of Inflammation Pharmacology of Inflammation Since inflammatory diseases and their therapy cover a broad range of scientific and medical fields, the encyclopedia will be co-edited by four international experts, a clinician and three researchers from the disciplines of immunology, biochemistry, and pharmacology, in this way providing students, basic and clinical scientists and practitioners in academia, hospitals and industry with valuable interlinked information. This living project will serve as a reliable and comprehensive data pool for everybody working in inflammation research. Owing to its dynamic nature, it will grow with time and future editions, becoming an indispensible source of information for academia, clinical practitioners and industry.
650		7	_2embne _aInflamación _9153270
700	1		_aParnham, Michael J _eeditor _4http://id.loc.gov/vocabulary/relators/edt _9100348
856	4	0	_uhttps://go.openathens.net/redirector/universidadeuropea.es?url=https://doi.org/10.1007/978-3-0348-0620-6 _zAcceso a este recurso digital (usuarios Universidad Europea de Madrid)
942			_2lcc _cLE
998			_aSI _cm _dz _feng _ggw _h0 _b12/2019 _ek _zSI