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Leucine-Rich Repeat kinase 2 (LRRK2) / Hardy J. Rideout, editor.

Contributor(s): Rideout, Hardy J.,, editor literario
Material type: materialTypeLabelE-bookSeries: (Advances in neurobiology ; volume 14).Publisher: Cham, Switzerland : Springer, [2017]Description: 1 recurso en línea.ISBN: 3319499696; 9783319499697.Subject: NeurogenéticaOnline resources: Acceso a este recurso digital (usuarios Universidad Europea de Madrid)Digital Resources Abstract: This is the first book to assemble the leading researchers in the field of LRRK2 biology and neurology and provide a snapshot of the current state of knowledge, encompassing all major aspects of its function and dysfunction. The contributors are experts in cell biology and physiology, neurobiology, and medicinal chemistry, bringing a multidisciplinary perspective on the gene and its role in disease. The book covers the identification of LRRK2 as a major contributor to the pathogenesis of Parkinson's Disease. It also discusses the current state of the field after a decade of research, putative normal physiological roles of LRRK2, and the various pathways that have been identified in the search for the mechanism(s) of its induction of neurodegeneration.
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Holdings
Item type Current library Collection Call number Status Date due Barcode Item holds
LIBRO-E NO PRÉSTAMO LIBRO-E NO PRÉSTAMO Madrid Digital Acceso Electrónico (UEM) FCBS | FCAD QP356.22 L483 2017 EB (Browse shelf(Opens below)) Acceso electrónico eBook.20023310
Total holds: 0

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This is the first book to assemble the leading researchers in the field of LRRK2 biology and neurology and provide a snapshot of the current state of knowledge, encompassing all major aspects of its function and dysfunction. The contributors are experts in cell biology and physiology, neurobiology, and medicinal chemistry, bringing a multidisciplinary perspective on the gene and its role in disease. The book covers the identification of LRRK2 as a major contributor to the pathogenesis of Parkinson's Disease. It also discusses the current state of the field after a decade of research, putative normal physiological roles of LRRK2, and the various pathways that have been identified in the search for the mechanism(s) of its induction of neurodegeneration.

Online resource; title from PDF title page (EBSCO, viewed April 05, 2017).

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