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Pathogenesis of Neuropathic Pain : Diagnosis and Treatment / edited by Daryl I. Smith, Hai Tran

Contributor(s): Smith, Daryl I, editor literario | Tran, Hai, editor literario
Material type: materialTypeLabelE-bookPublisher: Cham : Springer International Publishing, 2022Edition: First edition 2022.Description: 1 recurso en línea (XVII, 289 páginas) : 42 ilustraciones a color.ISBN: 9783030914554.Subject: Sistema nervioso -- Enfermedades -- Diagnóstico | Sistema nervioso -- Enfermedades -- Tratamiento | Dolor -- EtiologíaOnline resources: Acceso a este recurso digital (usuarios Universidad Europea de Madrid)Digital Resources
Contents:
Introduction -- Transitional Neuropathy: Chronification of Acute Pain -- Diabetic Neuropathy -- Complex Regional Pain Syndrome (CRPS) -- Autoimmune Disease-Related Neuropathy -- Methylation Disorder Neuropathy -- Alcohol-Related Neuropathy -- Perfusion-Related Neuropathy -- Uremic Neuropathy -- Chemotherapy Induced Neuropathy -- Antibiotics and Antifungals -- Miscellaneous -- Treatment -- Conclusion.
In: Springer Nature eBookSummary: This comprehensive source on the pathogenic origins of neuropathic pain covers the detailed molecular bases of the currently known neuropathies as classified by their pathogenic origins. Filling a critical need, this book fills the need for a resource on a syndrome that demands improved understanding by clinicians and researchers alike so that treatment options for patients are not categorically limited to a pill or a needle. If the clinician understands the origins of a patients' neuropathic pain, they can work cooperatively toward improving it with tailored therapies that don't create societal diseconomies and that ultimately are effective in helping patients. The book presents in detail the molecular bases of some currently known neuropathies by their pathogenetic origins, allowing clinicians to tailor more specific and more effective treatment regimens for their patients. For basic researchers, this book is a general resource to better direct research on neuropathy-specific molecular mechanisms. The improved understanding of the pathogenesis of neuropathic pain can then be used to develop more specific and more effective manipulations of these pathways.
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Holdings
Item type Current library Collection Call number Status Date due Barcode Item holds
LIBRO-E NO PRÉSTAMO LIBRO-E NO PRÉSTAMO Madrid Digital Acceso Electrónico (UEM) Ciencias de la Salud RB127 2022 ES (Browse shelf(Opens below)) Acceso electrónico eBook.24082135
Total holds: 0

Introduction -- Transitional Neuropathy: Chronification of Acute Pain -- Diabetic Neuropathy -- Complex Regional Pain Syndrome (CRPS) -- Autoimmune Disease-Related Neuropathy -- Methylation Disorder Neuropathy -- Alcohol-Related Neuropathy -- Perfusion-Related Neuropathy -- Uremic Neuropathy -- Chemotherapy Induced Neuropathy -- Antibiotics and Antifungals -- Miscellaneous -- Treatment -- Conclusion.

This comprehensive source on the pathogenic origins of neuropathic pain covers the detailed molecular bases of the currently known neuropathies as classified by their pathogenic origins. Filling a critical need, this book fills the need for a resource on a syndrome that demands improved understanding by clinicians and researchers alike so that treatment options for patients are not categorically limited to a pill or a needle. If the clinician understands the origins of a patients' neuropathic pain, they can work cooperatively toward improving it with tailored therapies that don't create societal diseconomies and that ultimately are effective in helping patients. The book presents in detail the molecular bases of some currently known neuropathies by their pathogenetic origins, allowing clinicians to tailor more specific and more effective treatment regimens for their patients. For basic researchers, this book is a general resource to better direct research on neuropathy-specific molecular mechanisms. The improved understanding of the pathogenesis of neuropathic pain can then be used to develop more specific and more effective manipulations of these pathways.

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