Image from Google Jackets

Gut Microbiota and Pathogenesis of Organ Injury / edited by Peng Chen

Contributor(s): Chen, Peng, editor | SpringerLink (Online service)
Material type: materialTypeLabelE-bookSeries: (Advances in Experimental Medicine and Biology, 0065-2598; 1238); (Biomedical and Life Sciences (Springer-11642)).Publisher: Singapore : Springer International Publishing, 2020Edition: First edition.Description: 1 recurso en línea (IX, 202 páginas) : 18 ilustraciones, 17 ilustraciones a color.ISBN: 9789811523854.Subject: Motilidad gastrointestinal -- Trastornos | Aparato digestivo | MicrobiologíaOnline resources: Acceso a este recurso digital (usuarios Universidad Europea de Madrid)Digital Resources
Contents:
Introduction -- Gut microbiota and alimentary tract injury -- Gut microbiota and liver injury - Acute liver injury -- Gut microbiota and liver injury - Chronic liver injury -- Gut microbiota and lung injury -- Gut microbiota and brain injury -- Gut microbiota and renal injury -- Gut microbiota and heart, vascular injury -- Gut microbiota and endocrine disorders -- Gut microbiota and immune responses -- Gut microbiota and multiple organ dysfunction syndrome (MODS).
Abstract: This book aims to introduce the latest research in gut microbiota by systematically summarizing how it modulates the pathogenesis of organ injury including alimentary tract injury, liver injury, lung injury, brain injury, renal injury, heart and vascular injury, endocrine disorders, immune responses and multiple organ dysfunction syndrome (MODS) during sepsis. Gut microbiota which is recognized as a new "organ" in the body has been demonstrated to be able to regulate the homeostasis of many organs. The key role played by gut microbiota is the hotspot in biomedical research nowadays. This book provides a state-of-the-art report on recent discoveries regarding the novel insight into the mechanisms of human diseases progression. It will also offer the overall picture of the pathophysiologic roles of gut microbiota. This book is helpful for graduate students and professional researchers to get the knowledge of frontiers in both gut microbiota and organ injury.
Tags from this library: No tags from this library for this title. Log in to add tags.
Star ratings
    Average rating: 0.0 (0 votes)
Holdings
Item type Current library Collection Call number Status Date due Barcode Item holds
LIBRO-E NO PRÉSTAMO LIBRO-E NO PRÉSTAMO Madrid Digital Acceso Electrónico (UEM) Ciencias de la Salud QR171.G29 2020 EB (Browse shelf(Opens below)) Acceso electrónico eBook.25062121
Total holds: 0

Introduction -- Gut microbiota and alimentary tract injury -- Gut microbiota and liver injury - Acute liver injury -- Gut microbiota and liver injury - Chronic liver injury -- Gut microbiota and lung injury -- Gut microbiota and brain injury -- Gut microbiota and renal injury -- Gut microbiota and heart, vascular injury -- Gut microbiota and endocrine disorders -- Gut microbiota and immune responses -- Gut microbiota and multiple organ dysfunction syndrome (MODS).

This book aims to introduce the latest research in gut microbiota by systematically summarizing how it modulates the pathogenesis of organ injury including alimentary tract injury, liver injury, lung injury, brain injury, renal injury, heart and vascular injury, endocrine disorders, immune responses and multiple organ dysfunction syndrome (MODS) during sepsis. Gut microbiota which is recognized as a new "organ" in the body has been demonstrated to be able to regulate the homeostasis of many organs. The key role played by gut microbiota is the hotspot in biomedical research nowadays. This book provides a state-of-the-art report on recent discoveries regarding the novel insight into the mechanisms of human diseases progression. It will also offer the overall picture of the pathophysiologic roles of gut microbiota. This book is helpful for graduate students and professional researchers to get the knowledge of frontiers in both gut microbiota and organ injury.

There are no comments on this title.

to post a comment.
Share